Physiotherapists in Redcar treat many different conditions including the issues profiled below.

Damage To The Spine By Bending Forward

Injuries sustained in bending occur when the forces involved exceed the coping capacity of the tissues of the spinal structures. The movements of the lumbar spinal bones in a living person undergoing a rapid forward bend are controlled mostly by the strength and length of the disc and ligamentous tissues. Initial damage occurs to the more superficial ligaments which connect the spines of the individual vertebrae posteriorly and if some side bending or twisting motion is added then the capsules of the facet joints can be stretched to their limits. The rear part of the disc wall may act like a ligament in extreme bend, restricting further bending.

In slower bending where a position is held for a time the resistance of the spinal tissues to bending decreases, allowing greater movements between the vertebrae and a slow change in the internal disc alignment. Due to the mechanical properties of the tissues quick movements have an increased likelihood of damaging the ligaments and discs, while holding the spine in flexion for some time may both reduce the protection activity of the back muscles and the ability of the ligaments to protect the lumbar segments as they move.

Activity and Muscle Strength

When a person starts to train for strength or power they start exerting high forces for short periods of time, i.e. they use heavier weights for smaller numbers of repetitions. This causes large changes in the fast twitch type 2 muscles fibres, increasing their size but not changing their number. This process is known as mechanically induced hypertrophy and results from the muscle cells synthesising increased amounts of muscle proteins to build into larger muscle. Muscles can also add new sections to the areas where they join the tendon if they are subjected to stretching forces.

In contrast, if muscles are inactive they reduce the making of muscle proteins within a few hours and the muscle starts to atrophy, to lose size and strength. In injuries if a muscle is immobilised in a shortened position both its size and its length will be reduced, with non-contracting substances becoming more dominant. This leads to a muscle which is shorter, more stiff and less easily lengthened, a significantly negative outcome in that immobilisation for fractures will have unwanted effects on the local muscular structures. Modern methods concentrate on immobilising a joint only if essential and only for the shortest necessary period.